<?xml version="1.0" encoding="UTF-8"?><rss xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:content="http://purl.org/rss/1.0/modules/content/" xmlns:atom="http://www.w3.org/2005/Atom" version="2.0"><channel><title><![CDATA[Cellular senescence is a biological stress response where cells cease dividing but do not die, instead lingering in tissue and emitting a toxic cocktail of inflammatory signals.]]></title><description><![CDATA[<p>Cellular senescence is a biological stress response where cells cease dividing but do not die, instead lingering in tissue and emitting a toxic cocktail of inflammatory signals. In the liver, immune cells known as macrophages can enter this "zombie" state, continuously accumulating and driving the chronic inflammation associated with both aging and fatty liver disease.<br /><a href="https://mastodon.social/tags/CellBiology" rel="tag">#<span>CellBiology</span></a> <a href="https://mastodon.social/tags/Immunology" rel="tag">#<span>Immunology</span></a> <a href="https://mastodon.social/tags/Hepatology" rel="tag">#<span>Hepatology</span></a> <a href="https://mastodon.social/tags/Gerontology" rel="tag">#<span>Gerontology</span></a> <a href="https://mastodon.social/tags/MolecularBiology" rel="tag">#<span>MolecularBiology</span></a> <a href="https://mastodon.social/tags/sflorg" rel="tag">#<span>sflorg</span></a><br /><a href="https://www.sflorg.com/2026/04/cbio04162602.html" rel="nofollow noopener"><span>https://www.</span><span>sflorg.com/2026/04/cbio0416260</span><span>2.html</span></a></p>]]></description><link>https://board.circlewithadot.net/topic/e79550d5-a632-4de8-b1ce-686a7c34f713/cellular-senescence-is-a-biological-stress-response-where-cells-cease-dividing-but-do-not-die-instead-lingering-in-tissue-and-emitting-a-toxic-cocktail-of-inflammatory-signals.</link><generator>RSS for Node</generator><lastBuildDate>Fri, 01 May 2026 12:37:06 GMT</lastBuildDate><atom:link href="https://board.circlewithadot.net/topic/e79550d5-a632-4de8-b1ce-686a7c34f713.rss" rel="self" type="application/rss+xml"/><pubDate>Thu, 16 Apr 2026 12:04:13 GMT</pubDate><ttl>60</ttl></channel></rss>