<?xml version="1.0" encoding="UTF-8"?><rss xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:content="http://purl.org/rss/1.0/modules/content/" xmlns:atom="http://www.w3.org/2005/Atom" version="2.0"><channel><title><![CDATA[Topics tagged with immunology]]></title><description><![CDATA[A list of topics that have been tagged with immunology]]></description><link>https://board.circlewithadot.net/tags/immunology</link><generator>RSS for Node</generator><lastBuildDate>Fri, 01 May 2026 12:34:09 GMT</lastBuildDate><atom:link href="https://board.circlewithadot.net/tags/immunology.rss" rel="self" type="application/rss+xml"/><pubDate>Invalid Date</pubDate><ttl>60</ttl><item><title><![CDATA[Polμ (Polymerase mu) is a crucial protein that facilitates DNA repair in macrophages during an immune response, ensuring the survival of these essential cells.]]></title><description><![CDATA[Polμ (Polymerase mu) is a crucial protein that facilitates DNA repair in macrophages during an immune response, ensuring the survival of these essential cells. By protecting innate immune cells from the genetic damage caused by their own pathogen-destroying mechanisms, Polμ enables effective tissue repair and limits chronic inflammation. #Immunology #MolecularBiology #Physiology #sflorghttps://www.sflorg.com/2026/04/imgy04272601.html]]></description><link>https://board.circlewithadot.net/topic/0cbbe515-e05d-416b-ab31-8509123b5323/polμ-polymerase-mu-is-a-crucial-protein-that-facilitates-dna-repair-in-macrophages-during-an-immune-response-ensuring-the-survival-of-these-essential-cells.</link><guid isPermaLink="true">https://board.circlewithadot.net/topic/0cbbe515-e05d-416b-ab31-8509123b5323/polμ-polymerase-mu-is-a-crucial-protein-that-facilitates-dna-repair-in-macrophages-during-an-immune-response-ensuring-the-survival-of-these-essential-cells.</guid><dc:creator><![CDATA[sflorg@mastodon.social]]></dc:creator><pubDate>Invalid Date</pubDate></item><item><title><![CDATA[Cellular senescence is a biological stress response where cells cease dividing but do not die, instead lingering in tissue and emitting a toxic cocktail of inflammatory signals.]]></title><description><![CDATA[Cellular senescence is a biological stress response where cells cease dividing but do not die, instead lingering in tissue and emitting a toxic cocktail of inflammatory signals. In the liver, immune cells known as macrophages can enter this "zombie" state, continuously accumulating and driving the chronic inflammation associated with both aging and fatty liver disease.#CellBiology #Immunology #Hepatology #Gerontology #MolecularBiology #sflorghttps://www.sflorg.com/2026/04/cbio04162602.html]]></description><link>https://board.circlewithadot.net/topic/e79550d5-a632-4de8-b1ce-686a7c34f713/cellular-senescence-is-a-biological-stress-response-where-cells-cease-dividing-but-do-not-die-instead-lingering-in-tissue-and-emitting-a-toxic-cocktail-of-inflammatory-signals.</link><guid isPermaLink="true">https://board.circlewithadot.net/topic/e79550d5-a632-4de8-b1ce-686a7c34f713/cellular-senescence-is-a-biological-stress-response-where-cells-cease-dividing-but-do-not-die-instead-lingering-in-tissue-and-emitting-a-toxic-cocktail-of-inflammatory-signals.</guid><dc:creator><![CDATA[sflorg@mastodon.social]]></dc:creator><pubDate>Invalid Date</pubDate></item></channel></rss>